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nikon fn 1 upright microscope  (Nikon)


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    Nikon nikon fn 1 upright microscope
    Nikon Fn 1 Upright Microscope, supplied by Nikon, used in various techniques. Bioz Stars score: 97/100, based on 1178 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/upright+fn+1+nikon+microscope/ECLIPSE+FN1/pm40379709-336-8-8
    Average 97 stars, based on 1178 article reviews
    nikon fn 1 upright microscope - by Bioz Stars, 2026-09
    97/100 stars

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    Related Articles

    Microscopy:

    Article Title: Activation and polarization of striatal microglia and astrocytes are involved in bradykinesia and allodynia in early-stage parkinsonian mice
    Article Snippet: .. SR-101-labeled astrocytes were identified using near-infrared DIC and fluorescent illumination under an upright FN-1 Nikon microscope equipped with a CCD camera (Flash 4.0 LTE, Hamamatsu, Japan) and a 40 × water-immersion objective (WD, 3.5 mm; NA, 0.80). ..

    Article Title: Subthalamic neurons interact with nigral dopaminergic neurons to regulate movement in mice.
    Article Snippet: © 2023 Scandinavian Physiological Society.. Published by John Wiley & Sons Ltd 1School of basic medical sciences, Xuzhou Medical University, Xuzhou, China 2Institute of Nervous System Diseases, Xuzhou Medical University, Xuzhou, China 3School of Anesthesiology, Xuzhou Medical University, Xuzhou, China 4Department of Anesthesiology, Drum Tower Hospital, affiliated to Nanjing University, Nanjing, China 5Jiangsu Province Key Laboratory in Anesthesiology, Xuzhou Medical University, Xuzhou, China



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    Image Search Results


    A , heat map representations and Ca 2+ signals illustrating high K + ‐evoked Ca 2+ activity under control conditions after the removal of extracellular Ca 2+ and after inhibition of L‐type Ca 2+ channels using nimodipine (10 µM). Scale bar = 50 µm. Raw Ca 2+ signals were deconvolved into fast and slow F / F 0 signal components, and traces from all cells are shown overlaid. In each trace the bold black line is the averaged Ca 2+ signals from all cells. B–D , summarized data showing the percentage of cells activated by a high K + PSS (physiological salt solution), the mean amplitude of fast Ca 2+ responses ( C ) and the mean amplitude of slow, persistent Ca 2+ responses ( D ). Statistical significance ( P < 0.05) using repeated‐measures one‐way ANOVA with the Geisser–Greenhouse correction and Dunnett's multiple comparisons test ( n = 5 animals in each experiment vs . control).

    Journal: The Journal of Physiology

    Article Title: Mitochondria regulate inositol triphosphate‐mediated Ca 2+ release triggered by voltage‐dependent Ca 2+ entry in resistance arteries

    doi: 10.1113/JP288022

    Figure Lengend Snippet: A , heat map representations and Ca 2+ signals illustrating high K + ‐evoked Ca 2+ activity under control conditions after the removal of extracellular Ca 2+ and after inhibition of L‐type Ca 2+ channels using nimodipine (10 µM). Scale bar = 50 µm. Raw Ca 2+ signals were deconvolved into fast and slow F / F 0 signal components, and traces from all cells are shown overlaid. In each trace the bold black line is the averaged Ca 2+ signals from all cells. B–D , summarized data showing the percentage of cells activated by a high K + PSS (physiological salt solution), the mean amplitude of fast Ca 2+ responses ( C ) and the mean amplitude of slow, persistent Ca 2+ responses ( D ). Statistical significance ( P < 0.05) using repeated‐measures one‐way ANOVA with the Geisser–Greenhouse correction and Dunnett's multiple comparisons test ( n = 5 animals in each experiment vs . control).

    Article Snippet: Images of Ca 2+ signals and immunostaining were obtained using a Nikon FN‐1 (Amstelveen, Netherlands) upright fluorescence microscope equipped with a 40× (0.8 numerical aperture) or 16× (0.8 numerical aperture) water dipping lens.

    Techniques: Activity Assay, Control, Inhibition

    A , top, heat map representation of basal (left) and high K + ‐evoked (right) Ca 2+ activity in the absence and presence of the phospholipase C blocker, U73122 (2 µM). Bottom, raw Ca 2+ signals from each cell shown in the heat map images. B and C , summarized data showing the mean amplitude ( B ) of high K + PSS‐evoked Ca 2+ signals and the percentage of cells responding to high K + PSS ( C ). Statistical significance was determined using a repeated‐measured two‐way ANOVA with the Geisser–Greenhouse correction and uncorrected Fisher's LSD (least significant difference) for multiple comparisons (treatment vs . control for basal activity, treatment vs . control for high K + ‐evoked activity; n = 5 animals).

    Journal: The Journal of Physiology

    Article Title: Mitochondria regulate inositol triphosphate‐mediated Ca 2+ release triggered by voltage‐dependent Ca 2+ entry in resistance arteries

    doi: 10.1113/JP288022

    Figure Lengend Snippet: A , top, heat map representation of basal (left) and high K + ‐evoked (right) Ca 2+ activity in the absence and presence of the phospholipase C blocker, U73122 (2 µM). Bottom, raw Ca 2+ signals from each cell shown in the heat map images. B and C , summarized data showing the mean amplitude ( B ) of high K + PSS‐evoked Ca 2+ signals and the percentage of cells responding to high K + PSS ( C ). Statistical significance was determined using a repeated‐measured two‐way ANOVA with the Geisser–Greenhouse correction and uncorrected Fisher's LSD (least significant difference) for multiple comparisons (treatment vs . control for basal activity, treatment vs . control for high K + ‐evoked activity; n = 5 animals).

    Article Snippet: Images of Ca 2+ signals and immunostaining were obtained using a Nikon FN‐1 (Amstelveen, Netherlands) upright fluorescence microscope equipped with a 40× (0.8 numerical aperture) or 16× (0.8 numerical aperture) water dipping lens.

    Techniques: Activity Assay, Control

    A , top, heat map images of Ca 2+ signals in response to photolysis of caged IP 3 in arteries that have been denuded of endothelial cells. Dashed green circle: area of photolysis. Scale bar = 50 µm. Bottom, Ca 2+ traces from the single cells activated in the top panel. The bold red line is the averaged Ca 2+ signals from cells in the uncaging area. B , summary data of average amplitude of Ca 2+ peaks; ( C ) propagation area of Ca 2+ signals after photolysis. Statistical significance ( P < 0.05) determined using the Friedman test with Dunn's multiple comparisons test ( n = 5 animals).

    Journal: The Journal of Physiology

    Article Title: Mitochondria regulate inositol triphosphate‐mediated Ca 2+ release triggered by voltage‐dependent Ca 2+ entry in resistance arteries

    doi: 10.1113/JP288022

    Figure Lengend Snippet: A , top, heat map images of Ca 2+ signals in response to photolysis of caged IP 3 in arteries that have been denuded of endothelial cells. Dashed green circle: area of photolysis. Scale bar = 50 µm. Bottom, Ca 2+ traces from the single cells activated in the top panel. The bold red line is the averaged Ca 2+ signals from cells in the uncaging area. B , summary data of average amplitude of Ca 2+ peaks; ( C ) propagation area of Ca 2+ signals after photolysis. Statistical significance ( P < 0.05) determined using the Friedman test with Dunn's multiple comparisons test ( n = 5 animals).

    Article Snippet: Images of Ca 2+ signals and immunostaining were obtained using a Nikon FN‐1 (Amstelveen, Netherlands) upright fluorescence microscope equipped with a 40× (0.8 numerical aperture) or 16× (0.8 numerical aperture) water dipping lens.

    Techniques:

    Depolarization of the plasma membrane induces Ca 2+ influx via L‐type VDCCs that produces a steady elevation in Ca 2+ , which activates IP 3 Rs to evoke repetitive Ca 2+ release events from the intracellular Ca 2+ store. Depolarization of the mitochondrial membrane potential reduces IP 3 R activity. Similarly inhibition of the ATP synthase increases ROS production, which also decreases IP 3 R activity. Ca 2+ entry via VDCC is unaffected by changes in the mitochondrial membrane potential or inhibition of the ATP synthase. IP 3 Rs, inositol phosphate receptors; ROS, reactive oxygen species; RyRs, ryanodine receptors; SR, sarcoplasmic reticulum; VDCC, voltage‐dependent Ca 2+ channel.

    Journal: The Journal of Physiology

    Article Title: Mitochondria regulate inositol triphosphate‐mediated Ca 2+ release triggered by voltage‐dependent Ca 2+ entry in resistance arteries

    doi: 10.1113/JP288022

    Figure Lengend Snippet: Depolarization of the plasma membrane induces Ca 2+ influx via L‐type VDCCs that produces a steady elevation in Ca 2+ , which activates IP 3 Rs to evoke repetitive Ca 2+ release events from the intracellular Ca 2+ store. Depolarization of the mitochondrial membrane potential reduces IP 3 R activity. Similarly inhibition of the ATP synthase increases ROS production, which also decreases IP 3 R activity. Ca 2+ entry via VDCC is unaffected by changes in the mitochondrial membrane potential or inhibition of the ATP synthase. IP 3 Rs, inositol phosphate receptors; ROS, reactive oxygen species; RyRs, ryanodine receptors; SR, sarcoplasmic reticulum; VDCC, voltage‐dependent Ca 2+ channel.

    Article Snippet: Images of Ca 2+ signals and immunostaining were obtained using a Nikon FN‐1 (Amstelveen, Netherlands) upright fluorescence microscope equipped with a 40× (0.8 numerical aperture) or 16× (0.8 numerical aperture) water dipping lens.

    Techniques: Clinical Proteomics, Membrane, Activity Assay, Inhibition